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Vascular endothelial cells and myosin: Mechanism of molecular motor-myosins in vascular endothelial cells

Submission Deadline: 21 December 2023 Submit to Special Issue

Guest Editors

Dr. Yanni Lv
Department of Pharmacy, The First Affiliated Hospital of Nanchang University, Jiangxi, China
Interests: endothelial niche; endothelial biology; traditional Chinese medicine; exosome; neuroinflammation

Dr. Yisong Qian
Institute of Translational Medicine, Nanchang University, Nanchang 330001, China.
Interests: Endothelial biology; sepsis; viral infectious diseases; macrophage activation; signal transduction

Dr. Menghao Huang
Institute of Translational Medicine, Nanchang University, Nanchang 330001, China.
Interests: Endothelial biology; viral infectious diseases; alcoholic liver disease; lipid metabolism; signal transduction


Endothelial cells, forming the inner wall of the blood vessels, participated in the body's pathological and physiological processes of immune, tumor, infection, etc. The most important component of molecular motor is myosin. Phosphorylation of myosin promotes its binding to F-actin, is thought to play a crucial role in the regulation of muscle contraction. Myosin was first discovered in muscle tissue. After 1970, many myosins were gradually discovered. There are 24 members in the superfamily.

However, the current literature focused on review studies. This topic tried deeply to study the knowledge basis of myosin on endothelial cells. The aim of this Special Issue is to encourage submissions, including original research and review articles, that contribute innovative knowledge to further our understanding of the signaling and mechanisms of myosins in vascular endothelial cells.

Potential topics keywords include but are not limited to the following:

Animal, cellular, and molecular myosins of vascular endothelial cells response to nervous system disease/neuroinflammation

Discovery of novel myosin biomarkers and mechanisms related to the vascular endothelial cells

Signaling and mechanisms of tissue damage caused by myosin dysfunction

Compounds that act on vascular endothelial cells dysfunction via intervention of myosins

Inhibition mechanism of myosins induced cell apoptosis, pyroptosis, and necrosis on vascular endothelial cells


vascular endothelial cells; myosin; molecular motor; molecular mechanism

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